Update citedin in frontmatter .md files

wikipathways · Sep 14, 2024 · 0070042 · 0070042
1 parent d0999d3
commit 0070042
Show file tree

Hide file tree

Showing 10 changed files with 34 additions and 1 deletion.
diff --git a/pathways/WP236/WP236.md b/pathways/WP236/WP236.md
@@ -48,6 +48,9 @@ citedin:
  title: In silico transcriptional analysis of asymptomatic and severe COVID-19 patients
  reveals the susceptibility of severe patients to other comorbidities and non-viral
  pathological conditions (2023)
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 description: The different classess of factors involved in adipogenesis are shown.
  Adipogenesis is the process by which fat cells differentiate from preadipocytes
  to adipocytes (fat cells). Adipose tissue, composed of white and brown adipose tissue,

diff --git a/pathways/WP2525/WP2525.md b/pathways/WP2525/WP2525.md
@@ -26,6 +26,9 @@ citedin:
 - link: 10.1159/000535120
  title: Human Monocytes Exposed to SARS-CoV-2 Display Features of Innate Immune Memory
  Producing High Levels of CXCL10 upon Restimulation (2023)
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 communities: []
 description: This pathway shows the (one) carbon metabolism over grey and related
  pathways around it. The one carbon donor pathway (WP2190) has been added to this

diff --git a/pathways/WP3297/WP3297.md b/pathways/WP3297/WP3297.md
@@ -31,6 +31,9 @@ citedin:
 - link: 10.1186/s40001-024-01951-z
  title: 'Non-dialyzable uremic toxins and renal tubular cell damage in CKD patients:
  a systems biology approach (2024)'
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 communities:
 - ExRNA
 description: 'Summary of reported extracellular vesicle (EV) release from cardiac

diff --git a/pathways/WP3527/WP3527.md b/pathways/WP3527/WP3527.md
@@ -17,6 +17,10 @@ authors:
 - Fehrhart
 - Marvin M2
 - Eweitz
+citedin:
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 description: 'The source of this pathway is RNA-Seq data from single-cell pre-implantation
  embryos, Supplemental Table 1 from [http://www.nature.com/nsmb/journal/v20/n9/full/nsmb.2660.html
  Yan et al.] Analysis options for running the single-cell analysis workflow in AltAnalyze

diff --git a/pathways/WP3995/WP3995.md b/pathways/WP3995/WP3995.md
@@ -26,6 +26,9 @@ citedin:
  title: Bioinformatics and system biology approach to identify the influences of
  SARS-CoV-2 infections to idiopathic pulmonary fibrosis and chronic obstructive
  pulmonary disease patients (2021)
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 communities:
 - Diseases
 - RareDiseases

diff --git a/pathways/WP4149/WP4149.md b/pathways/WP4149/WP4149.md
@@ -22,6 +22,9 @@ citedin:
 - link: 10.3390/pathogens12111373
  title: Transcriptional Profiling of SARS-CoV-2-Infected Calu-3 Cells Reveals Immune-Related
  Signaling Pathways (2024)
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 description: The transcription factors involved in white fat cell differentiation
  are shown, specifically their pattern of activity from the preadipocyte to the adipocyte
  (fat cells) stage. Adipose tissue, composed of white and brown adipose tissue, is

diff --git a/pathways/WP4191/WP4191.md b/pathways/WP4191/WP4191.md
@@ -23,6 +23,9 @@ citedin:
 - link: 10.1097/MD.0000000000033917
  title: Exploring the pharmacological mechanism of Duhuo Jisheng Decoction in treating
  intervertebral disc degeneration based on network pharmacology (2023)
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 description: 'Caloric restriction leads to a decrease in the ATP/AMP ratio, thereby
  activating the key nutrient sensor of the body: AMPK. AMPK then blocks mTOR function,
  as mTOR has an important role in regulating the balance between cell growth and

diff --git a/pathways/WP4688/WP4688.md b/pathways/WP4688/WP4688.md
@@ -29,6 +29,10 @@ authors:
 - Eweitz
 - Fehrhart
 - Finterly
+citedin:
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 communities:
 - IEM
 - Lipids

diff --git a/pathways/WP4904/WP4904.md b/pathways/WP4904/WP4904.md
@@ -12,6 +12,10 @@ authors:
 - Egonw
 - Fehrhart
 - Eweitz
+citedin:
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 communities:
 - COVID19
 description: A recent GWAS study was tweeted (https://twitter.com/BrentRichards19/status/1251550109931888643)

diff --git a/pathways/WP678/WP678.md b/pathways/WP678/WP678.md
@@ -20,7 +20,10 @@ authors:
 - DeSl
 - Eweitz
 - Egonw
-citedin: ''
+citedin:
+- link: 10.1038/s41598-023-33585-2
+ title: Bioinformatics analysis of the pathogenic link between Epstein-Barr virus
+ infection, systemic lupus erythematosus and diffuse large B cell lymphoma (2023)
 communities: []
 description: The cytochrome P450-dependent formation of polyunsaturated fatty acid
  epoxides is an important biochemical pathway creating mediators of inflammation